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Hb Shelby + Beta0-thalassemia Codon 39

Hb Leslie

, 

Hb Deaconess

Profile card
Created on 2026/09/08

General information

Globin chain involved

Beta-globin
Beta⁰-thal.

Status

Compound Heterozygous

Migration zones

No Zone
Z8

Migration positions

166

Sickle Cell Disease: No

Thalassemic variant: No

Capillary Electrophoresis

Fractions

Value %

Hb Shelby

89.7

Hb F

2.3

Hb A2

6.8

Denatured Hb

1.2

Comments

The combination of the mildly unstable Hb Shelby variant with Codon 39 Beta0-thalassemia leads to an additional denatured fraction in Z1 and the complete absence of Hb A. The specific pattern of this profile prevents the software from displaying the different zones.

Mutation data

Compound Heterozygous Hb Shelby

Mutation

HGVS Nomenclature

Beta 131(H9) Gln>Lys

HBB:c.394C>A

In combination with

Beta0-Thalassemia Codon 39

Mutation

HGVS Nomenclature

Beta 39(C5) Gln>Stop

HBB:c.118C>T

Hematological parameters

Name

Result

RBC Count

Low

Total Hemoglobin

Low

MCV

Low

MCH

Low

Blood smear

Increased target cells

Other analysis

Low hematocrit

Comments on hematology

Microcytosis, reticulocytosis

Clinical context

Clinical presentation

Mild hemolytic anemia symptoms, moderate splenomegaly

Clinical risk

May induce intermediate anemia when associated with Beta0-thalassemia, but without consequence when associated with Hb S

Variant information

Stability

Mildly unstable

Oxygen affinity

Slightly decreased

Ethnicities in literature

Found in Black & Caucasian populations: met in a few Black families living in the USA, in some members of a Sardinian family, in a Turkish patient, in a Brazilian citizen, in a 29-year-old pregnant female from Czech Republic origin living in the UK, and in a 47-year-old female born in Bolivia and living in Spain

Comments on variant information

The rare variant Hb Shelby was also reported in compound heterozygosity with Hb S, Hb C, beta-thalassemia and in double heterozygosity with alpha-thalassemia.

Hb Shelby inhibits Hb S polymerization when associated with this common variant.

Scientific Literature

Filters

  • Hb A2'

    ,

    Hb B2

  • Hb A2-Babinga

  • Hb A2-Coburg

  • Hb Abruzzo

  • Hb Ahvaz

  • Hb Andrew-Minneapolis

  • Hb Ann Arbor

  • Hb Arta + Beta0-thalassemia Codon 39

  • Hb Arya

  • Hb Aubagne

  • Hb Bassett

  • Hb Beograd

    ,

    Hb D-Camperdown

  • Hb Bougardirey-Mali

  • Hb Brem-sur-Mer

  • Hb Brest

  • Hb British Columbia

  • Hb Bruxelles

  • Hb C (heterozygous)

  • Hb C (homozygous - no zone)

  • Hb C (homozygous - with zones)

  • Hb Camperdown

  • Hb Chad

    ,

    Hb E-Keelung

  • Hb C-Harlem

    ,

    Hb C-Georgetown

  • Hb C-Harlem + Alpha+-thalassemia

    ,

    Hb C-Georgetown

  • Hb Cocody

No hemoglobin profiles found

Migration zones

Migration position

Single value (1-300) or range (e.g., 20-35)

Mutation types

Sickle Cell Disease

New Case & Recently Updated